Immunobiology – superb science built upon a totally flawed assumption. Part Four – Methinks they do protest too much

Flicking through the book, or settled down reading great chunks of it, one picks up several undercurrents. One is, as I have mentioned, a Jenneric influence in support of Him, the Almighty Vaccinologist and Noble Saviour of All. But there is also a protection system of their very own. Yes, they do have an awareness of a core deep mistrust of their endeavours and they undertake manoeuvres in attempts to outflank possible criticisms.

Everybody with any objectivity can look at the huge rise in allergic type disorders and agree that their increase has followed modern medical use of attempting to artificially induce in the population a “protection” against infection through the process of vaccination with antigens in a soup of adjuvanting chemicals, designed to over-ride normal physiological processing of the antigenic materials and create a B-cell and T-cell memory, as described in previous sections.

That they might be uneasy about this is supported by the fact that there are three chapters, 11, 12 &13, dealing with the issues. I’ll focus on 12 – “Allergy and hypersensitivity” but it is clear that they have no intension of addressing the elephant in these chapters – not even a hint that vaccines’ collateral damaging effects include the allergic spectrum of disorders.

P517: “ Allergy occurs when an individual who has produced IgE in response to an innocuous antigen or allergen subsequently encounters the same [substance]” Then “There are two main components of the immune response leading to IgE production [names, but only NAMES them] and the fate of the [B lymphocyte] is also impacted by the antigen, the dose of antigen and the route of presentation.”

“As many as 40% of people in Western populations show exaggerated tendency to mount IgE responses to a wide variety of common environmental allergens. This state is called Atopy and has a strong familial basis.” Well, yes, with 40% of the population atopic that is EVERY family!

On genetics: “ Studies of atopic families have identified regions of chromosomes 11q and 5q that seem to be important in atopy; candidate genes that could affect IgE are present in these regions.” And I really want to quote their genetic evidences – for sheer opacity and meaninglessness. Hey, well I suffered reading through it!

But then they give in anyway and admit (P524) “The prevalence of atopic allergy, and of asthma in particular, is increasing in economically advanced regions of the World, an observation that is best explained by environmental factors”. An increase of 0 to 40% in thirty years cannot readily be pinned on genetics!

So, what are their EFs?

“The four main candidates are:

  1. Changes in exposure to infectious diseases in early childhood
  2. Environmental pollution
  3. Allergen levels
  4. Dietary changes

“Of these 1 is most plausible at present. This known as the “hygiene hypothesis”

They waver on it but categorically say 3 and 4 are no use and 2 could go either way. 1 is, of course, so untrue as you cannot remove bacteria, virus and fungi from any lived in environment on the planet. But it gives them a distractor story.

Mostly they just get lost in jargon, running around naming things and thinking this shows that they understand and are in control of the facts. It doesn’t and they are not! This is SO typical:

P521 “ TH2 cells produce a characteristic, coordinated array of cytokines and chemokines that maintain and amplify the polarisation of the response and stimulate class switching of B-cells to IgE production. The cytokines include IL-4, IL-5, IL-9, IL-10 and IL-13. Cytokines IL-4 and IL-13 activate the Janus family tyrosine kinases Jak-1 and Jak-3 which ultimately leads to the phosphorylation of the Transcriptional Regulator Stat6 present in T and B lymphocytes. Mice lacking functional IL-4, IL-13 or Stat6 have impaired TH2 responses and impaired IgE switching demonstrating the key importance of these cytokines and their signalling pathways.”

Mice, you notice! They use a lot of mice. They’re a great tool for examining allergy. Hey, that’s what they say. Personally I’ve never seen a mouse with a skin rash but that’s just me. Asthmatic mice?

P528 “Most IgE is cell bound and engages effector mechanisms of the immune system by different pathways from other antibody isotypes.” This is to say that this antibody causes the body to undergo many of the profounder symptoms of allergy – rash, temperature, mucus production, coughing, sneezing and the like. Gives you flu, really!

About greencentre

Non grant supported hence independent scientist, green activist, writer and forest planter.
This entry was posted in Allergy, Antibodies, Antigen, Atopic individuals, B-cells, Bacteria, Chromosomal flaws?, Ecology of disease, Flu, Genetic studies, Immunobiology, Infectious diseases, Scientific method, T-cells, Vaccination, Vaccine damage. Bookmark the permalink.

Leave a Reply

Fill in your details below or click an icon to log in: Logo

You are commenting using your account. Log Out /  Change )

Google photo

You are commenting using your Google account. Log Out /  Change )

Twitter picture

You are commenting using your Twitter account. Log Out /  Change )

Facebook photo

You are commenting using your Facebook account. Log Out /  Change )

Connecting to %s